incidence<\/span> in premature babies, especially those with very low birth weight, and lesions can develop where adhesives such as for monitoring leads have been attached.<\/p>\nAnetoderma seems to be the most commonly reported in Central Europe.<\/p>\n
There have been rare cases of anetoderma affecting family members, including monozygotic (identical) twins.<\/p>\n
There are two forms of anetoderma: primary<\/span> and secondary. The cause of the primary form is unknown.<\/p>\nSecondary anetoderma occurs at the site of other unrelated skin conditions including:<\/p>\n
\n- Infections: chickenpox, syphilis\n<\/li>\n
- Tumors: pilomatrixoma, plasmacytoma, schwannoma, cutaneous<\/span> Cell B lymphoma<\/span>, youth<\/span> xanthogranuloma\n<\/li>\n
- Childish hemangioma<\/span><\/li>\n
- \nInflammatory<\/span> skin conditions: acne, granuloma<\/span> annulare, deep lupus\n<\/li>\n<\/ul>\n
Both forms of anetoderma can be associated with systemic<\/span> diseases including:<\/p>\n\n- Infections: Lyme disease (Borrelia), leprosy, HIV<\/span> infection<\/span>\n<\/li>\n
- Inflammatory\/autoimmune<\/span> conditions: systemic lupus erythematosus (SLE), Graves disease, Addison's disease, Sj\u00f6gren syndrome<\/span>,<\/li>\n
- Medications - penicillamine<\/li>\n<\/ul>\n
There appears to be a particular association with the presence of antiphospholipids antibodies<\/span>, often without the other features necessary to diagnose antiphospholipid syndrome.<\/p>\nThe old classification of the Schweninger-Buzzi and Jadassohn-Pellizari types, non-inflammatory versus inflammatory, is no longer considered useful as both types of injury<\/span> can be seen in a patient and many clinically non-inflammatory lesions show inflammation<\/span> in histology<\/span>. This classification does not predict forecast<\/span> and therefore it is no longer used.<\/p>\nMany theories have been proposed to describe the mechanism of development of anetoderma, but at this time this remains unknown.<\/p>\n
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<\/span>Clinical characteristics of anetoderma.<\/span><\/h2>\nThe typical clinical presentation of anetoderma is multi-turn. well defined<\/span> 1\u20132 cm lesions with wrinkled skin covering a palpable<\/span> depression, the so-called buttonhole sign<\/span>.<\/p>\nLesions may be flat, raised, or depressed, skin-colored, or bluish-white.<\/p>\n
Anetoderma lesions are most commonly found on the chest, back, neck, and arms, but anywhere can be affected. They are not related to hair<\/span> follicles<\/span>.<\/p>\n\n<\/span>Anetoderma<\/span><\/h5>\n<\/section>\n\n\n
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Chicken pox scars<\/p>\n
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Deep lupus<\/p>\n<\/p><\/div>\n<\/section>\n
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<\/span>How is anetoderma diagnosed?<\/span><\/h2>\nAnetoderma should be considered clinically for pathologist<\/span> knows how to make special spots on the skin biopsy<\/span> These specimens are needed to demonstrate the loss of elastic fibers<\/span> in the upper to middle dermis. In routine spots, the skin looks relatively normal, perhaps with some signs of inflammation.<\/p>\nSome authors suggest doing blood tests for Borrelia serology<\/span> where appropriate, and for autoimmune conditions, including antiphospholipid antibodies (lupus anticoagulant and anticardiolipin).<\/p>\n<\/span>What is the treatment for anetoderma?<\/span><\/h2>\nThere is no successful treatment to reverse the already formed anetoderma lesions. Individual lesions can be excised. When an underlying condition has been diagnosed, successful treatment can prevent further lesions from forming.<\/p>\n<\/section>","protected":false},"excerpt":{"rendered":"
\u00bfQu\u00e9 es la anetodermia? La anetodermia es una condici\u00f3n poco com\u00fan en la cual el tejido el\u00e1stico en el dermis se pierde, lo que resulta en una depresi\u00f3n en la…<\/p>","protected":false},"author":8,"featured_media":0,"menu_order":0,"comment_status":"closed","ping_status":"closed","template":"","format":"standard","meta":{"footnotes":""},"categories":[204],"tags":[],"class_list":["post-6946","wiki","type-wiki","status-publish","format-standard","category-glosario-definiciones"],"_links":{"self":[{"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/wiki\/6946","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/wiki"}],"about":[{"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/types\/wiki"}],"author":[{"embeddable":true,"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/users\/8"}],"replies":[{"embeddable":true,"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/comments?post=6946"}],"version-history":[{"count":0,"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/wiki\/6946\/revisions"}],"wp:attachment":[{"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/media?parent=6946"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/categories?post=6946"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/doctorhoogstra.com\/en\/wp-json\/wp\/v2\/tags?post=6946"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}